Lipopolysaccharides from Salmonella Minnesota, Alexa Fluor™ 488 conjugate
Citations & References (6)
Invitrogen™
Lipopolysaccharides from Salmonella Minnesota, Alexa Fluor™ 488 conjugate
This bright, green-fluorescent Alexa Fluor 488 conjugate can be used to follow lipopolysaccharide binding, transport and cell-internalization processes.Read more
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Catalog Number
Quantity
L23356
100 μg
Catalog number L23356
Price (CNY)
2,977.00
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Ends: 31-Dec-2025
3,931.00
Save 954.00 (24%)
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Quantity:
100 μg
Price (CNY)
2,977.00
飞享价
Ends: 31-Dec-2025
3,931.00
Save 954.00 (24%)
Each
Add to cart
This bright, green-fluorescent Alexa Fluor 488 conjugate can be used to follow lipopolysaccharide binding, transport and cell-internalization processes.
For Research Use Only. Not for use in diagnostic procedures.
Specifications
Detection MethodFluorescence
Dye TypeAlexa Fluor Dyes
Excitation/Emission495/519
Quantity100 μg
Shipping ConditionRoom Temperature
Product LineAlexa Fluor
Product TypeLipopolysaccharide Conjugate
Unit SizeEach
Contents & Storage
Store in freezer (-5 to -30°C) and protect from light.
Citations & References (6)
Citations & References
Abstract
Surfactant protein A directly interacts with TLR4 and MD-2 and regulates inflammatory cellular response. Importance of supratrimeric oligomerization.
'The purpose of the current study was to examine the binding of pulmonary surfactant protein A (SP-A) to TLR4 and MD-2, which are critical signaling receptors for lipopolysaccharides (LPSs). The direct binding of SP-A to the recombinant soluble form of extracellular TLR4 domain (sTLR4) and MD-2 was detected using solid-phase ... More
Activation of innate immunity in the CNS triggers neurodegeneration through a Toll-like receptor 4-dependent pathway.
Innate immunity is an evolutionarily ancient system that provides organisms with immediately available defense mechanisms through recognition of pathogen-associated molecular patterns. We show that in the CNS, specific activation of innate immunity through a Toll-like receptor 4 (TLR4)-dependent pathway leads to neurodegeneration. We identify microglia as the major lipopolysaccharide (LPS)-responsive ... More
Lipopolysaccharide (LPS) labeled with Alexa 488 hydrazide as a novel probe for LPS binding studies.
Authors:Triantafilou K, Triantafilou M, Fernandez N
Journal:Cytometry
PubMed ID:11084617
BACKGROUND: Lipopolysaccharide (LPS) comprises the outer cell wall of all gram-negative bacteria. It consists of an oligosaccharide core and lipid A. All LPS-induced biological responses are lipid A-dependent. Once released, LPS triggers a host systemic inflammatory response that leads to septic shock. Binding studies have helped to reveal some of ... More
The toll-like receptor TLR4 is necessary for lipopolysaccharide-induced oligodendrocyte injury in the CNS.
The immediate or innate immune response is the first line of defense against diverse microbial pathogens and requires the expression of recently discovered toll-like receptors (TLRs). TLR4 serves as a specific receptor for lipopolysaccharide (LPS) and is localized on the surface of a subset of mammalian cells. Although innate immunity ... More
Lipopolysaccharide Is Cleared from the Circulation by Hepatocytes via the Low Density Lipoprotein Receptor.