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Invitrogen
COL22A1 Polyclonal Antibody
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COL22A1 Antibody (PA5-70815) in WB
Aging 2023 -
Figure 7 COL22A1 knockdown inhibits glioma cell proliferation and migration. ( A ) RT-qPCR was used to detect the expression of COL22A1, IGF2BP2, and MPO in NHA, U87, LN229, and LN18 cells. ( B ) IHC was used to detect the expression of COL22A1 in grade 4 diffuse gliomas and adjacent non-tumor tissues. ( C ) Western blotting was used to detect the expression of COL22A1 in negative control (NC) and COL22A1 k... View More
Please note: We are reviewing Western blot images included in the antibody testing data in our catalog, including those provided by third parties. Unless expressly labeled or annotated as “raw-unedited”, Western blot images included in the antibody testing data in our catalog may have been edited, optimized or otherwise adjusted for presentation.
产品信息
PA5-70815
应用
建议稀释比
已发表文章
产品规格
种属反应
Human
已发表种属
Human
宿主/亚型
Rabbit
/ IgG
分类
Polyclonal
类型
Antibody
抗原
synthetic peptide directed towards the N-terminal region of human COL22A1
偶联物
Unconjugated
Unconjugated
Unconjugated
形式
Liquid
浓度
0.5 mg/mL
规格
50 µg
纯化类型
Affinity chromatography
保存液
PBS with 2% sucrose
内含物
0.09% sodium azide
保存条件
-20°C, Avoid Freeze/Thaw Cycles
运输条件
Ambient (domestic); Wet ice (international)
RRID
靶标信息
This gene encodes member of the collagen family which is thought to contribute to the stabilization of myotendinous junctions and strengthen skeletal muscle attachments during contractile activity. It belongs to the fibril-associated collagens with interrupted triple helix (FACIT) subset of the collagen superfamily, which associate with collagen fibers through their C-terminal collagenous domains and mediate protein-protein interactions through their N-terminal noncollagenous domains. The encoded protein is deposited in the basement membrane zone of the myotendinous junction which is present only at the tissue junctions of muscles, tendons, the heart, articular cartilage, and skin. A knockdown of the orthologous zebrafish gene induces a muscular dystrophy by disruption of the myotendinous junction.
仅用于科研。不用于诊断过程。未经明确授权不得转售。
生物信息学
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