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Invitrogen
DUSP10 Polyclonal Antibody
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DUSP10 Antibody (PA5-109665) in WB
Western blot analysis of DUSP10 in extracts of BT-474 cells. Samples were incubated with DUSP10 Polyclonal antibody (Product # PA5-109665) using a dilution of 1:1,000, followed by HRP Goat Anti-Rabbit IgG (H+L) at a dilution of 1:10,000. Lysates/proteins: 25 µg per lane. Blocking buffer: 3% nonfat dry milk in TBST. Detection: ECL Basic Kit. Exposure time: 90s.
产品信息
PA5-109665
产品规格
种属反应
Human,
Mouse,
Rat
宿主/亚型
Rabbit
/ IgG
分类
Polyclonal
类型
Antibody
抗原
Recombinant fusion protein containing a sequence corresponding to amino acids 1-300 of human DUSP10 (NP_009138.1).
偶联物
形式
Liquid
浓度
0.75 mg/mL
纯化类型
Affinity Chromatography
保存液
PBS, pH 7.3, with 50% glycerol
内含物
0.02% sodium azide
保存条件
-20°C, Avoid Freeze/Thaw Cycles
运输条件
Ambient (domestic); Wet ice (international)
RRID
产品详细信息
Immunogen sequence: MPPSPLDDRV VVALSRPVRP QDLNLCLDSS YLGSANPGSN SHPPVIATTV VSLKAANLTY MPSSSGSARS LNCGCSSASC CTVATYDKDN QAQTQAIAAG TTTTAIGTST TCPANQMVNN NENTGSLSPS SGVGSPVSGT PKQLASIKII YPNDLAKKMT KCSKSHLPSQ GPVIIDCRPF MEYNKSHIQG AVHINCADKI SRRRLQQGKI TVLDLISCRE GKDSFKRIFS KEIIVYDENT NEPSRVMPSQ PLHIVLESLK REGKEPLVLK GGLSSFKQNH ENLCDNSLQL QECREVGGGA
靶标信息
Microsomal long and very long chain fatty acid elongation uses malonyl-CoA as the 2-carbon donor and consists of 4 sequential reactions. GPSN2 catalyzes the final step, reducing trans-2, 3-enoyl-CoA to saturated acyl-CoA. Microsomal long and very long chain fatty acid elongation uses malonyl-CoA as the 2-carbon donor and consists of 4 sequential reactions. TER catalyzes the final step, reducing trans-2, 3-enoyl-CoA to saturated acyl-CoA.
仅用于科研。不用于诊断过程。未经明确授权不得转售。
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生物信息学
蛋白别名: dual specificity phosphatase MKP-5; Dual specificity protein phosphatase 10; MAP kinase phosphatase 5; Mitogen-activated protein kinase phosphatase 5; serine/threonine specific protein phosphatase
基因别名: 2610306G15Rik; AI158871; DUSP10; MKP-5; MKP5
UniProt ID: (Human) Q9Y6W6, (Mouse) Q9ESS0
Entrez Gene ID: (Human) 11221, (Mouse) 63953, (Rat) 63995
inactivation of MAPK activity
regulation of adaptive immune response
protein dephosphorylation
response to stress
JNK cascade
dephosphorylation
response to lipopolysaccharide
peptidyl-tyrosine dephosphorylation
negative regulation of JUN kinase activity
negative regulation of protein kinase activity by regulation of protein phosphorylation
oligodendrocyte differentiation
negative regulation of oligodendrocyte differentiation
negative regulation of respiratory burst involved in inflammatory response
regulation of brown fat cell differentiation
negative regulation of stress-activated MAPK cascade
regulation of innate immune response
negative regulation of JNK cascade
response to organic substance
