MEM 非必需氨基酸溶液 (100X)
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MEM 非必需氨基酸溶液 (100X)
Gibco™

MEM 非必需氨基酸溶液 (100X)

Gibco™ MEM 非必须氨基酸用作细胞培养基的添加剂,可以提高细胞生长和活性。Gibco® MEM 非必需氨基酸含有与标准最小必需培养基 (MEM) 中相同的 100X 浓度非必需氨基酸了解更多信息
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货号数量
11140050100 mL
1114007620 x 100 mL
货号 11140050
价格(CNY)
439.00
Each
添加至购物车
数量:
100 mL
价格(CNY)
439.00
Each
添加至购物车
Gibco™ MEM 非必须氨基酸用作细胞培养基的添加剂,可以提高细胞生长和活性。Gibco® MEM 非必需氨基酸含有与标准最小必需培养基 (MEM) 中相同的 100X 浓度非必需氨基酸。完整的配方请见此处。

两工厂 cGMP 生产和质量体系
Gibco™ MEM 非必需氨基酸溶液在位于纽约格兰德岛的符合 cGMP 要求的工厂内生产。该工厂是在 FDA 注册的医疗器械生产商,通过 ISO 13485 标准认证。为确保供应链连续性,我们同时提供由我们的苏格兰工厂生产的相同的 Gibco® MEM 非必需氨基酸溶液产品 (11140-035)。后者亦是在FDA登记的医疗仪器生产商,且符合ISO 13485标准。
仅用于研究和生产用途。不可用于临床诊断或直接用于人类或动物。
规格
细胞类型哺乳动物细胞
最大浓度100 X
生产质量cGMP-compliant under the ISO 13485 standard
数量100 mL
有效期18 个月
运输条件室温
形式液体
产品类型非必需氨基酸
无菌无菌过滤
pH0.9
Unit SizeEach
内容与储存
储存在 2–8°C 下。避光。

常见问题解答 (FAQ)

何为Gibco MEM非必需氨基酸?

MEM非必需氨基酸是一种细胞培养添加剂,能够促进细胞生长和存活。MEM非必需氨基酸产品以100倍浓度形式为用户提供与标准MEM培养基相同的非必需氨基酸组份。用户可点击此处(https://www.thermofisher.com/us/en/home/technical-resources/media-formulation.165.html)查阅其完整配方。

Where can I find the formulation of MEM Non-Essential Amino Acids Solution (100X) (Cat. No. 11140050)?

The formulation of MEM Non-Essential Amino Acids Solution (100X) can be found at this link: https://www.thermofisher.com/us/en/home/technical-resources/media-formulation.165.html

Find additional tips, troubleshooting help, and resources within our Cell Culture Support Center.

What is the solvent used for MEM Non-Essential Amino Acids Solution (100X)?

The solvent is distilled water.

Find additional tips, troubleshooting help, and resources within our Cell Culture Support Center.

What is Gibco MEM Non-Essential Amino Acids?

MEM Non-Essential Amino Acids are used as a supplement for cell culture medium, to increase cell growth and viability. MEM Non-Essential Amino Acids contains the same non-essential amino acids found in the standard Minimum Essential Medium (MEM) at a strength of 100X. The complete formulation is available here (http://www.thermofisher.com/us/en/home/technical-resources/media-formulation.165.html).

Find additional tips, troubleshooting help, and resources within our Cell Culture Support Center.

引用和文献 (12)

引用和文献
Abstract
Identification of a novel redox-sensitive gene, Id3, which mediates angiotensin II-induced cell growth.
Authors:Mueller Cornelius; Baudler Stephanie; Welzel Hilke; Böhm Michael; Nickenig Georg;
Journal:Circulation
PubMed ID:12021231
BACKGROUND: Reactive oxygen species, such as superoxide (O(2)(-)), are involved in the abnormal growth of various cell types. Angiotensin II (Ang II) is one of the most potent inducers of oxidative stress in the vasculature. The molecular events involved in Ang II-induced proliferation of vascular smooth muscle cells (VSMCs) are ... More
Patient-derived glioblastoma organoids as real-time avatars for assessing responses to clinical CAR-T cell therapy
Authors:Meghan Logun, Xin Wang, Yusha Sun, Stephen J Bagley, Nannan Li, Arati Desai, Daniel Y Zhang, MacLean P Nasrallah, Emily Ling-Lin Pai, Bike Su Oner, Gabriela Plesa, Donald Siegel, Zev A Binder, Guo-Li Ming, Hongjun Song, Donald M O'Rourke
Journal:Cell Stem Cell
PubMed ID:39657679
Patient-derived tumor organoids have been leveraged for disease modeling and preclinical studies but rarely applied in real time to aid with interpretation of patient treatment responses in clinics. We recently demonstrated early efficacy signals in a first-in-human, phase 1 study of dual-targeting chimeric antigen receptor (CAR)-T cells (EGFR-IL13Rα2 CAR-T cells) ... More
Effects of an indole derivative on cell proliferation, transfection, and alternative splicing in production of lentiviral vectors by transient co-transfection.
Authors:Mier NC,Roper DK
Journal:PloS one
PubMed ID:38833479
Lentiviral vectors derived from human immunodeficiency virus type I are widely used to deliver functional gene copies to mammalian cells for research and gene therapies. Post-transcriptional splicing of lentiviral vector transgene in transduced host and transfected producer cells presents barriers to widespread application of lentiviral vector-based therapies. The present study ... More
Mutation of the cytoplasmic domain of the integrin beta 3 subunit. Differential effects on cell spreading, recruitment to adhesion plaques, endocytosis, and phagocytosis.
Authors: Ylänne J; Huuskonen J; O'Toole T E; Ginsberg M H; Virtanen I; Gahmberg C G;
Journal:J Biol Chem
PubMed ID:7721884
'The cytoplasmic domain of the beta subunit of the alpha IIb beta 3 integrin is required for cell spreading on fibrinogen. Here we report that deletion of six amino acids from the COOH terminus of the beta 3 (I757TYRGT) totally abolished cell spreading and formation of adhesion plaques, whereas retaining ... More
Gene correction of the apolipoprotein (Apo) E2 phenotype to wild-type ApoE3 by in situ chimeraplasty.
Authors:Tagalakis AD, Graham IR, Riddell DR, Dickson JG, Owen JS,
Journal:J Biol Chem
PubMed ID:11278248
'Apolipoprotein (apo) E is a polymorphic plasma protein, synthesized mainly by liver. Here, we evaluate whether synthetic DNA-RNA oligonucleotides (chimeraplasts) can convert a dysfunctional isoform, apoE2 (C --> T, R158C), which causes Type III hyperlipidemia and premature atherosclerosis, into apoE3. First, we treated recombinant Chinese hamster ovary cells stably secreting ... More