RPMI 1640 培养基最初是为了悬浮培养人白血病单层细胞而开发的。Roswell Park Memorial Institute (RPMI) 1640 培养基被发现适用于多种哺乳动物细胞,包括了解更多信息
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11835030
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11835055
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货号 11835030
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500 mL
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价格(CNY)
425.80
飞享价
Ends: 31-Dec-2025
543.00
共减 117.20 (22%)
Each
添加至购物车
RPMI 1640 培养基最初是为了悬浮培养人白血病单层细胞而开发的。Roswell Park Memorial Institute (RPMI) 1640 培养基被发现适用于多种哺乳动物细胞,包括 HeLa 细胞、Jurkat 细胞、 MCF-7 细胞、PC12 细胞、PBMC 细胞、星形胶质细胞和癌细胞。针对广泛的细胞培养应用,我们提供多种组分的 RPMI 1640 改良培养基。使用培养基选择工具查找适合的配方。
Inhibition of both paracrine and autocrine VEGF/ VEGFR-2 signaling pathways is essential to induce long-term remission of xenotransplanted human leukemias.
Authors: Dias S; Hattori K; Heissig B; Zhu Z; Wu Y; Witte L; Hicklin D J; Tateno M; Bohlen P; Moore M A; Rafii S;
Journal:Proc Natl Acad Sci U S A
PubMed ID:11553814
'Antiangiogenic agents block the effects of tumor-derived angiogenic factors (paracrine factors), such as vascular endothelial growth factor (VEGF), on endothelial cells (EC), inhibiting the growth of solid tumors. However, whether inhibition of angiogenesis also may play a role in liquid tumors is not well established. We recently have shown that ... More
Regulation of neural differentiation by normal and mutant (G654A, amyloidogenic) gelsolin.
Authors: Westberg J A; Zhang K Z; Andersson L C;
Journal:FASEB J
PubMed ID:10463954
'Gelsolin belongs to a family of proteins that modulate the structural dynamics of cytoskeletal actin. Gelsolin activity is required for the redistribution of actin occurring during membrane ruffling, cell crawling, and platelet activation. A point mutation (G654A) in the gelsolin gene causes a dominantly inherited systemic amyloidosis called familial amyloidosis ... More
Inhibition of mitochondrial respiration by endogenous nitric oxide: a critical step in Fas signaling.
We have found that activation of human adult T cell leukemia (Jurkat) cells with anti-Fas Ab leads, in a concentration-dependent manner, to an early burst of production of nitric oxide (NO), which inhibits cell respiration. This results in mitochondrial hyperpolarization, dependent on the hydrolysis of glycolytic ATP by the F1F(o)-ATPase ... More