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引用和文献 (15)
Invitrogen™
FluoroMyelin™ Green 荧光髓鞘染色剂 - 水溶液
FluoroMyelin™ Green 荧光髓鞘染色剂可通过单个20分钟标记步骤和洗涤，快速选择性地标记脑冷冻切片中的髓鞘。此染色剂可结合抗体和其他染料使用，并可结合标准组织化学方法处理冰冻切片材料。了解更多信息
| 货号 | 数量 |
|---|---|
| F34651 | 1 mL |
货号 F34651
价格（CNY)
4,613.00
Each
数量:
1 mL
价格（CNY)
4,613.00
Each
FluoroMyelin™ Green 荧光髓鞘染色剂可通过单个20分钟标记步骤和洗涤，快速选择性地标记脑冷冻切片中的髓鞘。此染色剂可结合抗体和其他染料使用，并可结合标准组织化学方法处理冰冻切片材料。
仅供科研使用。不可用于诊断程序。
规格
颜色绿色
检测方法荧光
适用于（设备）荧光显微镜
标签或染料FluoroMyelin™ Green
产品类型绿色荧光髓鞘染色
数量1 mL
运输条件室温
子细胞定位髓鞘
激发/发射479/598 nm
产品线FluoroMyelin
Unit SizeEach
内容与储存
室温避光储存。
常见问题解答 (FAQ)
我用了你们的FluoroMyelin染色剂，发现它会对胶质细胞之外的其他细胞染色。是不是产品有什么地方出错了？
FluoroMyelin染色剂可染色培养的神经元吗？
FluoroMyelin髓磷脂染色剂可染色石蜡切片吗？
FluoroMyelin髓磷脂染色剂是什么?
I used one of your FluoroMyelin stains and noticed it stains cells other than glial cells. Is there something wrong with the product?
引用和文献 (15)
引用和文献
Abstract
Akt1 deficiency affects neuronal morphology and predisposes to abnormalities in prefrontal cortex functioning.
Journal:Proc Natl Acad Sci U S A
PubMed ID:17077150
'There is accumulating evidence that AKT signaling plays a role in the pathogenesis of schizophrenia. We asked whether Akt1 deficiency in mice results in structural and functional abnormalities in prefrontal cortex (PFC). Exploratory transcriptional profiling revealed concerted alterations in the expression of PFC genes controlling synaptic function, neuronal development, myelination,
Inhibitor of apoptosis protein (IAP) profiling in experimental autoimmune encephalomyelitis (EAE) implicates increased XIAP in T lymphocytes.
Journal:J Neuroimmunol
PubMed ID:18055022
'In multiple sclerosis (MS) and its widely accepted animal model, experimental autoimmune encephalomyelitis (EAE), the failure of autoreactive immune cells to undergo apoptosis is thought to contribute to CNS tissue damage and disease progression. Promoting apoptosis of myelin-reactive immune cells in diseases such as MS, may delay disease progression and
Prion protein (PrPc) positively regulates neural precursor proliferation during developmental and adult mammalian neurogenesis.
Journal:Proc Natl Acad Sci U S A
PubMed ID:16492732
'The misfolding of the prion protein (PrP(c)) is a central event in prion diseases, yet the normal function of PrP(c) remains unknown. PrP(c) has putative roles in many cellular processes including signaling, survival, adhesion, and differentiation. Given the abundance of PrP(c) in the developing and mature mammalian CNS, we investigated
Neuronal glutathione deficiency and age-dependent neurodegeneration in the EAAC1 deficient mouse.
Journal:Nat Neurosci
PubMed ID:16311588
Uptake of the neurotransmitter glutamate is effected primarily by transporters expressed on astrocytes, and downregulation of these transporters leads to seizures and neuronal death. Neurons also express a glutamate transporter, termed excitatory amino acid carrier-1 (EAAC1), but the physiological function of this transporter remains uncertain. Here we report that genetically
Effect of chronic continuous or intermittent hypoxia and reoxygenation on cerebral capillary density and myelination.
Journal:Am J Physiol Regul Integr Comp Physiol
PubMed ID:16322350
Chronic hypoxia, whether continuous (CCH) or intermittent (CIH), occurs in many neonatal pathological conditions, such as bronchopulmonary dysplasia and obstructive sleep apnea. In this study, we explored the effect of CCH and CIH on cerebral capillary density and myelination. We subjected CD-1 mice starting at postnatal day 2 to either