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引用和文献 (7)
Invitrogen™
Pacific Orange™ 琥珀酰亚胺酯三乙基铵盐
Pacific Orange™ 琥珀酰亚胺酯是一种可被紫色激光 (405 nm) 激发的染料，最大发射波长 ∼551 nm。这使得它能够与其他可被紫色激光激发的荧光基团（如了解更多信息
| 货号 | 数量 |
|---|---|
| P30253 | 1 mg |
货号 P30253
价格（CNY)
5,250.00
Each
数量:
1 mg
价格（CNY)
5,250.00
Each
Pacific Orange™ 琥珀酰亚胺酯是一种可被紫色激光 (405 nm) 激发的染料，最大发射波长 ∼551 nm。这使得它能够与其他可被紫色激光激发的荧光基团（如 Pacific Blue™ 染料）配合使用，以应用于使用单一激发源进行的多参数分析。作为一类化合物，琥珀酰亚胺酯形式提供了非常有效和易用的反应化学试剂，以选择性地将染料与蛋白质、经修饰的核酸或其他生物分子上可及的伯胺基连接起来。琥珀酰亚胺酯是用于胺修饰的优良试剂，因为它们形成的共价键与蛋白质分子中连接氨基酸的肽键一样稳定。Pacific Orange™ 琥珀酰亚胺酯以1 mg 干粉制剂提供，应在 ≤-20°C 下干燥储存并避光。
仅供科研使用。不可用于诊断程序。
规格
化学反应性胺
发射551
激发405
标签或染料Pacific Orange™
产品类型琥珀酰亚胺酯
数量1 mg
反应一部分活性酯、琥珀酰亚胺酯
运输条件室温
标签类型Pacific 染料
产品线Pacific Orange
Unit SizeEach
内容与储存
储存在冰箱中 (-5 - -30°C) 并避光。
引用和文献 (7)
引用和文献
Abstract
Dual channel STED nanoscopy of lytic granules on actin filaments in natural killer cells.
Journal:Commun Integr Biol
PubMed ID:22808328
Natural killer (NK) cells are innate immune effectors that eliminate diseased and tumorigenic targets through the directed secretion of specialized secretory lysosomes, termed lytic granules. This directed secretion is triggered following the formation of an immunological synapse (IS), which is characterized by actin re-modeling and receptor organization at the interface
Tyramide signal amplification for analysis of kinase activity by intracellular flow cytometry.
Journal:Cytometry A
PubMed ID:20824632
'Intracellular flow cytometry permits quantitation of diverse molecular targets at the single-cell level. However, limitations in detection sensitivity inherently restrict the method, sometimes resulting in the inability to measure proteins of very low abundance or to differentiate cells expressing subtly different protein concentrations. To improve these measurements, an enzymatic amplification
Dual receptor T cells mediate pathologic alloreactivity in patients with acute graft-versus-host disease.
Journal:
PubMed ID:23740900
Acute graft-versus-host disease (aGVHD) results from a robust response of donor T cells transferred during hematopoietic stem cell transplantation (HSCT) to allogeneic peptide-major histocompatibility complex antigens. Previous investigations have not identified T cell subsets that selectively mediate either protective immunity or pathogenic alloreactivity. We demonstrate that the small subset of
Activation of T lymphocytes in atherosclerotic plaques.
Journal:Arterioscler Thromb Vasc Biol
PubMed ID:21960562
To decipher the immunologic mechanisms of plaque maturation and rupture, it is necessary to analyze the phenotypes and distribution of individual lymphocytes that migrate to the plaques, as well as their activation at different stages of plaque formation. We developed a protocol to isolate plaque-residing immune cells and analyze their
In vivo functional analysis and genetic modification of in vitro-derived mouse neutrophils.
Journal:FASEB J
PubMed ID:21368104
Mature neutrophils are notoriously short-lived immune cells that cannot be genetically manipulated. Analysis of gene function therefore requires genetically modified animals, which is expensive, time-consuming, and costly in animal life. Analysis of gene function in neutrophils in a physiologically relevant context thus represents a significant problem in the field. We